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Alzheimer’s disease (AD) represents the most prevalent form of dementia, progressively eroding memory, cognitive functions, and ultimately, the ability to carry out daily activities. Affecting millions globally, its neuropathological hallmarks include the accumulation of amyloid-beta plaques and neurofibrillary tangles composed of hyperphosphorylated tau protein, alongside significant neuroinflammation and oxidative stress. Current pharmaceutical interventions primarily offer symptomatic

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